||Mode of Action
||Acetaminophen/ Tylenol/ Paracetamol
||Restores depleted glutathione stores and protects athwart renal and hepatic want.
||Non-specific poisons bar cyanide, sinewy, lithium, caustics and alcohol.
||Absorption of garbage in the gastric and intestinal tracts. Interrupts the invadeo-hepatic cycle delay multiple dose.
|albuterol inhaler, insulin & glucose, NaHCO3, kayexalate
||Neuromuscular arrestade (paralytics)
|atropine sulfate or pralidoxime
||Competitive discountenance of muscarinic receptors.
||Amanita phalloides (Death cap plebeian)
||Not known; specific guard athwart sharp hepatic want; may dislodge amatoxin from protein-styptic topicalitys allowing extensiond renal excretion; may as-well hinder sharpness of amatoxin to hepatocytes.
||Rapidly many-sidedes delay fluoride ion.
||Deferoxamine acts by styptic unimpeded sinewy in the bloodstream and enhancing its encircleation in the urine.
digoxine immune fab
||Binds molecules of digoxin, making them secretive for styptic at their topicality of renewal on cells in the whole.
|dimercapol, edetate calcium, disodium,
||Chelation of frequented ions and endogenous metals (e.g., zinc, manganese, sinewy, copper).
||Extrapyramidal symptoms (EPS)
||A effective foe to acetylcholine in muscarinic receptors.
||Reverses the goods of benzodiazepines by competitive discountenance at the benzodiazepine styptic topicality on the GABAA receptor.
||A competitive hinderor of the enzyme alcohol dehydrogenase build in the liver. This enzyme plays a key role in the metabolism of ethylene glycol and methanol.
||Beta arresters and calcium agent arresters
||Stimulates the constituteation of adenyl cyclase causing intracellular extension in cycling AMP and augmentd glycogenolysis and rising serum glucose tension.
|Glucose (Dextrose 50%)
||Dextrose (the monosaccharide glucose) is used, exclusive and stored by whole tissues and is metabolized to carbon dioxide and impart delay the quit of immateriality.
||Reverses hypercoagulable recite by interacting delay antithrombin III. Used in structure delay vasodilator phentolamine or nitroprusside to hinder topical thrombosis and ischemia.
||Forms cyanocobalamin, a non-toxic metabolite that is easily excreted through the kidneys.
||Protects the salubrious cells from the goods of methotrexate conjuncture allowing methotrexate to invade and massacre cancer cells.
||A “chemoprotectant” garbage that reduces the undesired goods of undeniable chemotherapy garbages.
||Chemical submissive exact methemoglobinemia. Ifosamide-induced encephalopathy.
||Reduces methemoglobin to hemoglobin.
|nalmefene or naloxone
||Prevents or reverses the goods of opioids including respiratory depression, sedation and hypotension.
||Naloxone is believed to antagonize opioid goods by competing for the µ, κ and σ anodyne receptor topicalitys in the CNS, delay the principal harmony for the µ receptor.
||Anticholinesterase which causes hoard of acetylcholine at cholinergic receptor topicalitys.
|Nitrite, sodium and glycerytrinitrate
||Oxidizes hemoglobin to methemoglobin which binds the unimpeded cyanide and can augment endothelial cyanide detoxification by submissive vasodilation.
||Copper, gold, frequented, mercury, zinc, arsenic
||Chelation of metal ions.
||Regitine produces an alpha-adrenergic arrest of relatively inextensive term. It as-well has frequented, but near notable, decisive inotropic and chronotropic goods on cardiac muscle and vasodilator goods on vascular flatten muscle.
|phyostigmine or NaHCO3
||A mutable anticholinesterase which effectively extensions the tension of acetylcholine at the topicalitys of cholinergic transmission.
|Phytomenadione (Vitamin K.)
||Bypasses discountenance of Vitamin K epoxide reductase enzyme.
||Protamine that is strongly basic combines delay eageric heparin constituteing a stanch many-sided and neutralizes the anticoagulant disposition of twain garbages.
||Isoniazid, theophylline, monomethyl hydrazine. Adjunctive therapy in ethylene glycol poisoning.
||Reverses sharp pyridoxine want by promoting GABA structure. Promotes the transmutation of toxic metabolite glycolic eager to glycine.
||Neutralizes malignity by styptic delay circulating malignity components and delay topically deposited malignity by accumulating at the bite topicality.
||Prevents convertion of ferrous to ferric.
|Cardiotoxic garbage imposing steadsteadpay sodium agent (TCA, cocaine)
||Decreases harmony of cardiotoxic garbages to the steadsteadpay sodium agent.
||Promotes ionization of undecided eagers.
|Chlorine gas inhalational poisoning
||Neutralization of hydrochloric eager constituteed when chlorine gas reacts delay impart in the airways.
||Replenishes depleted thiosulphate stores by acting as sulfur donor needful for the transmutation of CN-O to thiocyanate through the renewal of sulfur transferase enzyme rhodanese.
||Alcohol, Wernicke-Korsakoff Syndrome
||Reverses sharp thiamine want
|Adjunctive in ethylene glycol
||Enhances detoxification of glyoxylic eager.
||Chemicals causing methemoglobinemia in patients delay G6PD want
||Reduces methemoglobin to hemoglobin.